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Too Much of A Good Thing: A Case of Suspected Acute Tubular Necrosis Provoked by Hypervitaminosis D

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MLA citation style (9th ed.)

Benavente K, Knox, M, and Peesapati, Meghna. Too Much of A Good Thing: A Case of Suspected Acute Tubular Necrosis Provoked by Hypervitaminosis D. . 2021. mushare.marian.edu/concern/generic_works/c95cd0f3-8306-4439-8bff-8bb58a520dcc?locale=es.

APA citation style (7th ed.)

B. K, K. M, & P. Meghna. (2021). Too Much of A Good Thing: A Case of Suspected Acute Tubular Necrosis Provoked by Hypervitaminosis D. https://mushare.marian.edu/concern/generic_works/c95cd0f3-8306-4439-8bff-8bb58a520dcc?locale=es

Chicago citation style (CMOS 17, author-date)

Benavente K, Knox, M, and Peesapati, Meghna. Too Much of A Good Thing: A Case of Suspected Acute Tubular Necrosis Provoked by Hypervitaminosis D. 2021. https://mushare.marian.edu/concern/generic_works/c95cd0f3-8306-4439-8bff-8bb58a520dcc?locale=es.

Note: These citations are programmatically generated and may be incomplete.

Calcitriol is a vitamin critical in regulating calcium
homeostasis, maintenance of musculoskeletal
integrity, and both a commonly prescribed medication
and over the counter supplement.
• However, the incidence of vitamin D toxicity is
escalating, manifesting clinically with confusion,
polyuria, polydipsia, muscle weakness, and nausea
and vomiting (1).
• While acute hypercalcemia, especially in the setting of
milk-alkali syndrome, has been demonstrated to
induce a reversible AKI, the direct cytotoxic effect of
excess Vitamin D on the renal parenchyma in the
setting of lower levels of serum calcium, has not been
well studied nor documented.

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