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Too Much of A Good Thing: A Case of Suspected Acute Tubular Necrosis Provoked by Hypervitaminosis D Public Deposited

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Benavente K, Knox, M, and Peesapati, Meghna. Too Much of A Good Thing: A Case of Suspected Acute Tubular Necrosis Provoked by Hypervitaminosis D. . 2021. https://mushare.marian.edu/concern/generic_works/c95cd0f3-8306-4439-8bff-8bb58a520dcc?locale=en

APA citation style

Benavente K, Knox, M, & Peesapati, Meghna. (2021). Too Much of A Good Thing: A Case of Suspected Acute Tubular Necrosis Provoked by Hypervitaminosis D. https://mushare.marian.edu/concern/generic_works/c95cd0f3-8306-4439-8bff-8bb58a520dcc?locale=en

Chicago citation style

Benavente K, Knox, M, and Peesapati, Meghna. Too Much of A Good Thing: A Case of Suspected Acute Tubular Necrosis Provoked by Hypervitaminosis D. 2021. https://mushare.marian.edu/concern/generic_works/c95cd0f3-8306-4439-8bff-8bb58a520dcc?locale=en

Note: These citations are programmatically generated and may be incomplete.

Calcitriol is a vitamin critical in regulating calcium homeostasis, maintenance of musculoskeletal integrity, and both a commonly prescribed medication and over the counter supplement. • However, the incidence of vitamin D toxicity is escalating, manifesting clinically with confusion, polyuria, polydipsia, muscle weakness, and nausea and vomiting (1). • While acute hypercalcemia, especially in the setting of milk-alkali syndrome, has been demonstrated to induce a reversible AKI, the direct cytotoxic effect of excess Vitamin D on the renal parenchyma in the setting of lower levels of serum calcium, has not been well studied nor documented.

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