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Enabling Study of Human Immune Dysfunction Using Yeast Gene 0ST3/UNGI

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MLA citation style (9th ed.)

Casey, Ariyanna. Enabling Study of Human Immune Dysfunction Using Yeast Gene 0st3/ungi. . 2018. mushare.marian.edu/concern/generic_works/2e45d6d0-fd09-468a-a833-ff1f517e82f3?locale=es.

APA citation style (7th ed.)

C. Ariyanna. (2018). Enabling Study of Human Immune Dysfunction Using Yeast Gene 0ST3/UNGI. https://mushare.marian.edu/concern/generic_works/2e45d6d0-fd09-468a-a833-ff1f517e82f3?locale=es

Chicago citation style (CMOS 17, author-date)

Casey, Ariyanna. Enabling Study of Human Immune Dysfunction Using Yeast Gene 0st3/ungi. 2018. https://mushare.marian.edu/concern/generic_works/2e45d6d0-fd09-468a-a833-ff1f517e82f3?locale=es.

Note: These citations are programmatically generated and may be incomplete.

OST3 is a yeast gene for Obligosaccharyl transferase, a homolog of the Magnesium Transport 1 gene in humans. This human gene produce XMEN disease. XMEN disease is an X-Linked immune system deficiency where the amount of CD4+ T cells present in the body are reduced in function. XMEN is caused by a mutation in the MAGT1 gene. The MAGT1 gene produces a protein called magnesium transport. As shown in Magnesium transport is important because it activates CD4+ T cells, allowing for infections to be effectively detected. A mutation in the MAGT1 gene, reduces the function and effectivity of CD4+ T cells. For the individual, this mutation would result in a higher risk of developing infection, pneumonia or cancer. The only known effective treatment for XMEN is stem cell therapy.

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  • Undergraduate Research Symposium

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