Enabling Study of Human Immune Dysfunction Using Yeast Gene 0ST3/UNGI
Público DepositedMLA citation style (9th ed.)
. 2018. mushare.marian.edu/concern/generic_works/2e45d6d0-fd09-468a-a833-ff1f517e82f3?locale=es. Enabling Study of Human Immune Dysfunction Using Yeast Gene 0st3/ungi.APA citation style (7th ed.)
(2018). Enabling Study of Human Immune Dysfunction Using Yeast Gene 0ST3/UNGI. https://mushare.marian.edu/concern/generic_works/2e45d6d0-fd09-468a-a833-ff1f517e82f3?locale=esChicago citation style (CMOS 17, author-date)
Enabling Study of Human Immune Dysfunction Using Yeast Gene 0st3/ungi. 2018. https://mushare.marian.edu/concern/generic_works/2e45d6d0-fd09-468a-a833-ff1f517e82f3?locale=es.Note: These citations are programmatically generated and may be incomplete.
OST3 is a yeast gene for Obligosaccharyl transferase, a homolog of the Magnesium Transport 1 gene in humans. This human gene produce XMEN disease. XMEN disease is an X-Linked immune system deficiency where the amount of CD4+ T cells present in the body are reduced in function. XMEN is caused by a mutation in the MAGT1 gene. The MAGT1 gene produces a protein called magnesium transport. As shown in Magnesium transport is important because it activates CD4+ T cells, allowing for infections to be effectively detected. A mutation in the MAGT1 gene, reduces the function and effectivity of CD4+ T cells. For the individual, this mutation would result in a higher risk of developing infection, pneumonia or cancer. The only known effective treatment for XMEN is stem cell therapy.
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EnablingStudyHumanImmuneDysfunctionCasey_2018.pdf | 2021-07-27 | Público | Descargar |