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Abnormal Trafficking of Endogenously Expressed BMPR2 Mutant Allelic Products in Patients with Heritable Pulmonary Arterial Hypertension
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BMP Signaling in Vascular Development and Disease.
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BMPR-II is Dispensable for Formation of the Limb Skeleton.
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Increased Susceptibility to Hypoxic Pulmonary Hypertension in Bmpr2 Mutant Mice is Associated with Endothelial Dysfunction in the Pulmonary Vasculature
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Loss of BMPR2 Leads to High Bone Mass Due to Increased Osteoblast Activity
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N-linked Glycosylation of the Bone Morphogenetic Protein Receptor Type 2 (BMPR2) Enhances Ligand Binding
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The Role of BMP2 Signaling in the Skeleton.
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